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angioedema swelling of lower layer of sin and underlying tissue. swelling may be in face, tongue, larynx, abdomen, arms and legs. often associated w/urticaria
compensated shock early shock where the body still maintains adequate perfusion
decompensated shock later shock where the body can no longer maintain adequate perfusion
Exsanguination severe bleeding, leading to death
hemorrhage excessive bleeding
irreversible shock stage of shock leading to inevitable death
mean arterial pressure(MAP) DBP+ 1/3(SBP-DBP)
Multiple Organ Dysfunction Syndrome (MODS) progressive failure of atleast two organ systems
Urticaria hives
3 causes of shocks pump problem(cardiac) pipes problem(vasodilation) fluid problem(hypovolemic)
compensated shock compensatory mechanisms increased HR and cardiac force of contraction increased vasoconstriction reduced urinary output to maintain intravascular volume
decompensated (progressive) shock falling or low BP are hallmark signs bodys defense mechanisms are no longer able to compensate for the decrease in cardiac output
signs of shock ALOC tachycardia progressing to absent pulses in decompensated shock pale,cool,clammy normal BP during compensated shock and falling BP in decompensating shock
s/s of compensated shock ALOC tachycardia pale, cool, clammy skin thirst normal BP
s/s of decompensated shock decreased LOC absent peripheral pulses mottling, cyanosis falling BP progressing to hypotension
shock management airway management control bleeding prevent heat loss rapid transport IV consider fluid bolus
s/s of neurogenic shock possible paralysis possible respiratory MOI indicative of probable spinal injury warm, flushed, dry skin hypotension even in early stage of shock slow pulse
s/s of anaphylactic shock skin: flushed,itching,urticaria,angioedema respiratory: dyspnea,wheezing,stridor,laryngospasm cardiovascular: widespread vasodilation,tachycardia
s/s of sepsis fever or hypothermia possible skin can be flushed, pale, or cyanotic ALOC dyspnea, abnormal lung sounds tachycardia, hypotension
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