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Microbiology and Immunology Practice Test Questions

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Microbiology and Immunology Practice Test Questions

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1. Canciclovir

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlAntigenic Drift: Small Point Mutations That Result In Loss Of Antibody Binding. Year To Year Reinfection With Influenza. Antigenic Shift: Rearrangements Of Rna Results In Completely New Virus. (Epidemic) Example: Influenza Due To Ha And Na VariationsMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

2. Initial Vent Setup Decisions

Antigenic Drift: Small Point Mutations That Result In Loss Of Antibody Binding. Year To Year Reinfection With Influenza. Antigenic Shift: Rearrangements Of Rna Results In Completely New Virus. (Epidemic) Example: Influenza Due To Ha And Na VariationsMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

3. Trpanosoma Gambiense & Rhodesiense

Antigenic Drift: Small Point Mutations That Result In Loss Of Antibody Binding. Year To Year Reinfection With Influenza. Antigenic Shift: Rearrangements Of Rna Results In Completely New Virus. (Epidemic) Example: Influenza Due To Ha And Na VariationsMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationIndication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual Control

4. Explain The Difference In Antigenic Shift And Antigenic Drift?

5. Rhabdoviridae: Rabies Virus

Mechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Www.Brain101.Info - 17 - â€¢ Structure: Ss (-) Rna, Helical Capsid, Enveloped, Bullet-shaped. - O Transcriptase, Rna-dependent Rna Polymerase Is Present. - â€¢ Replication: Cytoplasmic. - O Primary Transcription ------> Genome Replication ------> More Transcription And Translation Occur Post-replication. - O Nucleocapsids Bud Through Host Cell-membrane To Form New Particles. - â€¢ Epidemiology: - O Racoons Are The Most Common Animal Carrier Today. - O Bats Are Most Common Carrier In U.S. To Actually Result In Infection. - O Also Skunks, Wild Mammals, And Unvaccinated Domestic Mammals. - â€¢ Diagnosis: Look For Negri Bodies In The Animal Tissue. - â€¢ Manifestations: - O Pathogenesis: - Virus Replicates In Striated Muscle At Site Of Bite. Then It Travels Up Nerve Endings To Spinal Cord And - Brain. - Virus Hooks To Acetylcholine Receptors At Neuromuscular Junction. - O Symptoms: Fatal Encephalitis. Hypertonic Muscle Contraction, Convulsions, Coma, Death. - Hydrophobia Happens As Swallowing Would Precipitate A Spasm Of Throat Muscles. - â€¢ Treatment: No Treatment Known. - O Prevention: Post-exposure Immunization, Both Passive And Active, Should Be Given Prophylactically In - Cases Of Possible Infection. - Human Rabies Immune Globulin (Hrig) Administered On Same Day As First Vaccine Dose, But At A - Different Site. - Human Diploid Cell Vaccine (Hdcv) Is The New Active Vaccine (Inactivated Virus), Grown In Human - Diploid Fibroblast Cells. Given In 6 Doses. Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

6. Major Causes Of Neonatal Meningitis With Sepsis?

Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Listeria Monocytogenes, Streptococcus Agalactiae, And E. ColiIndication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual Control

7. Thrush:

Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlCandida Albicans

8. What Do Chlamydia Trachomatis Serotypes D-k Cause? (3)

Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Urethritis/ Pid - Neonatal Pneumonia -neonatal ConjuctivitisIndication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual Control

9. Trichinella Spiralis

10. Is Pneumocystis Carinii A Yeast?

Yes, But Originally Classified As A ProtozoaMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

11. What Kind Of Immunity (Antibody-mediated Or Cell Mediated) Is Involved In Autoimmunity?

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Antibody-mediated Immunity (B Cells)

12. What Bacteria Are Obligate Anaerobes?

13. A Vector In Which The Parasite Does Not Go Through Any Part Of Its Life Cycle During Transit. Ex: Fly With Bacteria On Feet, Lands On Food, You Later Ingest, Passing Bacteria To You

14. Path: Coccidioides Immitis:

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlSouthwest (Mexico), Dust, Amphotericin BMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

15. Name The Vaccines That Are Killed (4).

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration-rabies-influenza-hepatitis A-salk=KilledMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.

16. Where Do Enveloped Viruses Acquire Their Envelopes, And What Virus Is The Exception To This Rule?

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration-plasma Membrane-herpesviruses Which Acquire Their Envelope From The Nuclear MembraneMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.

17. Obstructive Diseases Like Copd Need What Kid Of I:E Times

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationShort I And Longer E

18. A Characteristic That Can Be Observed Or Felt Only By The Patient. Ex: Pain, Headache, Nausea

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlDisease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationSymptomMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.

19. Humoral Or Cell-mediated Immunity: Live Attenuated Vaccines

Both (With A Few Cases Of Reversion To Virulence)Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns PenetrationMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual Control

20. Grows Anywhere, Needs And Doesn't Need Oxygen. Ex: E.Coli And Staph

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlFaculative AnaerobeMechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

21. What Bug: Sepsis In A Newborn

22. Rickettsia Rickettsiae

Indication, Noninvasive/Invasive, Press/Volume, Partial/Full Support, Mode-ac, Simv W/ Or W/O Ps, Psv, Psv, Pcv, Dual ControlPoorly Staining Gram-negative Pleomorphic Rods. - â€¢ Family Properties: Rickettsiaceae - O Strict Intracellular Parasites - O Requirwe Nad And Atp For Growth - O Other Species: - Ehrlichia: Arthropod-vectored Infection Of Wbc's. No Rash. - Coxiella: Q-fever. Usually Carried By Farm Animals. - Res Involvement -- Hepatosplenomegaly. Maybe No Pneumonia. - No Rash. - â€¢ Epidemiology / At Risk: Found In The Eastern U.S., Spread By Ticks, In The Summer. - â€¢ Manifestations: Rocky Mountain Spotted Fever Is The Best Known Example. - O General Properties: - Arthropods Are Vectors. - They Infect Endothelial Cells, Starting With Small Capillaries And Working Their Way Up, Leading To - Thrombosis And Hemorrhage. - There Is A Prominent Rash And Fever. - O Time Course: - 4-7 Days: Extensive Rash, Extending From Extremities To Trunk. Rash Resembles Syphillus. - 2 Weeks: Very Sick. High Fever, Dic, Hypotension, Gram-negative Shock. - Cardiac Failure, Renal Failure, And Encephalitis Are Likely Causes Of Death. - O Other Rickettsial Infections: - Typhus: Several Species And Several Vectors. Rash Spreads From Trunk To Extremities, Rather Than Rsmf - Which Is The Opposite. - Rickettsial Pox: From Mites. Benign, Self-limited Rash. - â€¢ Processing: - O Specimen: Rash Biopsy For Rmsf. - O Culture: Egg Or Tissue Culture. - â€¢ Identification: - O Direct Fa On Endothelial Cells - O Serology: Indirect Fa To Measure Antibody Levels. - Antibody Titer Of 1:64, Or Acute:Convelescent Titer Of 4x - â€¢ Virulence: - O Outer Membrane Proteins - O Phospholipase: Acts As An Invasin, Helping Bugs Get Into Endothelial Cells. - O Lps: Strong Endotoxic Component. - O Actin Polymerization: Making Little Projection To Infect Other Cells. Some Sort Of Way For The Bugs To Spread From - Cell To Cell, Similar To Shigella. - â€¢ Host Immune Response: Probably Combination Of Antibody Plus Cell-mediated. Not Sure. - â€¢ Vaccine / Prevention: Protective Clothing. - â€¢ Treatment: Tetracycline. Mechanism - 5'-monophosphate Formed By A Cmv Viral Kinase Or Hsv/Vzv Thymidine Kinase. Cuanosine Analog. Triphosphate Formed By Cellular Kinases. Preferentially Inhibits Viral Dna Polymerase.
- Clinical Use - Cmv, Especially In Immunocompromised Patients.
- Toxicity - Leukopenia, Neutopenia, Thrombocytopenia, Renal Toxicity. More Toxic To Host Enzymes Than Acyclovir.
- Mechanism Of Resistance - Mutated Cmv Dna Polymerase Or Lack Of Viral Kinase.Disease - African Sleeping Sickness: Enlarged Lymph Nodes, Recurring Fever Due To Antigenic Variation, Somnolence, Coma
- Transmission - Tsetse Fly
- Diagnosis - Blood Smear
- Treatment - Suramin For Bloodborne Disease Or Melarsoprol For Cns Penetration

23. Spontvt

24. Reverse Transcriptase Inhibitors (Hiv)

25. Colonisation

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All trademarks, logos and brand names are the property of their respective owners. All company, product and service names used in this website are for identification purposes only. Use of these names, trademarks and brands does not imply endorsement.

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