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UK Medicine Practice Questions

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UK Medicine Practice Questions

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1. Chest-pain: What are the most common diagnosis of patients admitted to hospital with chest pain which is ruled not to be due to mi?

Mild disease needs no treatment. If symptomatic of platelets less than 20x10^9/l rx with prednisolone 1mg/kg/day90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

2. goutimport:-duplicate: What is the cause?

Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMild disease needs no treatment. If symptomatic of platelets less than 20x10^9/l rx with prednisolone 1mg/kg/day

3. Acute-coronary-syndrome: What is the long term management?

1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMild disease needs no treatment. If symptomatic of platelets less than 20x10^9/l rx with prednisolone 1mg/kg/dayMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

4. Immune-mediated-thrombocytopenic-purpura: What is the management?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMild disease needs no treatment. If symptomatic of platelets less than 20x10^9/l rx with prednisolone 1mg/kg/day1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

5. hypergammaglobulinaemiaimport:-duplicate: What are the causes?

Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)May be polyclonal or monoclonal. Causes of polyclonal include chronic major infection/disease e.g tb, sarcoid, chronic liver disease and autoimmune disease e.g ra causes of monoclonal include monoclonal gammopathy of unknown significance (mgus), multiple myeloma, waldenstroms macrogammaglobulinemia, heavy chain disease, leukaemia, lymphoma90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

6. Ecgs: What pattern do you get in a rbbb and why?

1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseRsr1 in v1 and deep s wave in v6. Remember by marrow (m shape in v1, w shape in v6, two r's reminding you it's a right bundle branch block.) R1 in v1 and deep s in v6 caused by late depolarization of right ventricle. (with a widened qrs complex!)

7. Aortic-stenosisimport:-duplicate duplicate:-front: What are the symptoms?

8. Autoantibodies-disease-associations: anti-smooth muscle antibody (sma)

Autoimmune hepatitis 70% pbc 50% idiopathic cirrhosis 30%Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease

9. The-cytochrome-p450-system: why is it so important?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Induction or inhibition of it affects drug metabolism. This may exacebate substrate toxicity or accelate drug metabolism. There is much genetic variation in the system.

10. Carbemazepineimport:-duplicate duplicate:-front: What are the adverse effects?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Rash, nausea, diplopia, dizziness, fluid retention, hyponaturaemia, blood dyscrasiasMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

11. Carcinoid: describe the classical carcinoid syndrome

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)Flushing, diarrhoea, wheezing, abdominal fibrosis, cardiac valvular fibrosis, stenosis and regurgitation.1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

12. Ethics&law;: What is the public health (control of diseases) act about?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)Public health, notifiable diseases, that sort of thing (1984)

13. Diabetes-mellitus: more concordance in twins in ti or tii

14. Hyperlipidemia: How do you make the diagnosis?

Do a blood test look at the patient first of course for clinical signs of hyperlipidemia e.g xanthelasma, corneal arcusMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

15. Pupil-disorders: horner's syndrome

Triad of ptosis, anhydrosis and miosis due to invasion of the sympathetic trunk90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

16. Ecgs: What is the significance of the upwards, downwards, and no, sloping of a depressed st segment?

Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Upsloping st depression is non-specific downsloping st depression suggests digoxin usage flat st depression suggest myocardial ischaemia

17. Hepatitis: What are the clinical features of hepatitis c?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMost acute infections are subclinical, jaundice occurs in only about 10% of cases. The majority of acute infections (over 70%) will develop into chronic persistant infections.1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

18. lymphodemaimport:-duplicate: What is it?

1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseAn abnormal collection of interstitial lymph fluid either due to congenital absence of the lymphatics (primary) or secondary to blockage of the lymphatics.Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

19. Morphine: is it a good analgesic for biliary colic?

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)No because it is associated with increased biliary motility and spasm and raises intra-biliary pressure.1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

20. sodium-cromoglycate-import:-duplicate: How does it work?

Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseNot known, thought to stabalise mast cells, prevents mast cell degranulation in vitro.

21. Coagulation: How do venous thrombi differ from arterial thrombi?

Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Appearance: red vs white content: fibrin rich vs platelet rich therapy: heparin and warfarin vs antiplatelet agents

22. Blood-tests: What does rdw stand for? What is its use? How is it calculated?

1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal diseaseRbc distribution width used to detect rbc of unequal sizes - anisocytosis = (mean rbc width + s.d rbc width) x 100 n.b it's seen in early fe-deficiency anaemia due to a haemorrhage as have rbs not lost in haemorrhage are normal size, new ones which are the product of fe-deficient erythropoiesis are small.Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)

23. Multiple-sclerosis: What are the typical signs and symptoms at presentation?

1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)Usually present with only one of: unilateral optic neuritis (pain on eye movement and rapid decline in central acuity), diplopia, ataxia. Other symptoms include autoniomic urge incontinance, constipation, impotence, uhthoffs phenomenon (visual deficits on excercise due to increased temp), vertigo, declining cognition, lhermittes symptom (electrical sensation in spine or limbs on neck flexion - also positive in cervical spondylitis and b12 deficiency) Monitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease

24. Abnormal-values: a platelet count of less than ____ requires treatment with predisolone

90% of cases primary 10% of cases secondary to increased nucleic acid turnover e.g leukaemia or reduced excreation of uric acid e.g chronic renal disease20x10^9/lMonitor for complications. Prophylaxis against thromboembolism, heparin, if large mi consider warfarin for 3/12 continue/start low dose aspirin start b-blocker. Continue ace-i or start one if evidence of hf. Start a statin. Address modifible risk factors (diet, smoking, excercise, treat hbp, diabetes and hyperlipedmia)1.Gastrooesophageal Disease (reflux, motility disorders, pud, gs) (approx 40%) 2.IHD (approx 30%) 3.Chest Wall syndromes (approx 30%)

25. Heart-sounds: What causes a loud pulmonary component of hsii

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❤ If you liked Fatskills, consider supporting us by checking out The Life Manuals You Never Got.

About | Explore | User Guide | Topics | Subjects | Doubt Solver | Career Aptitude Test | Answers | Free Tools | What Should We Know? Privacy | Terms |

Without work one finishes nothing. - Ralph Waldo Emerson© 2026 Fatskills.com

All trademarks, logos and brand names are the property of their respective owners. All company, product and service names used in this website are for identification purposes only. Use of these names, trademarks and brands does not imply endorsement.

About | Explore | User Guide | Topics | Subjects | Doubt Solver | Career Aptitude Test | Answers | Free Tools | What Should We Know?
Privacy | Terms |

Without work one finishes nothing. - Ralph Waldo Emerson
© 2026 Fatskills.com